Described novel regulator of protein inactive in over 50 percent of human tumors
Monday, July 21, 2014 - 11:00
in Health & Medicine
The interaction between HERC2 proteins with another protein called p53 is inactivated in more than half of human tumors, researchers have discovered. The study suggests that mutations in HERC2 also may be associated with cancer in humans. "In the laboratory we have observed that without HERC2 cells increases proliferation. It's the same effect as if they inactivated p53" says the lead author.