Herpes virus rearranges telomeres to improve viral replication

An infection with herpes simplex virus 1 causes rearrangements in telomeres, small stretches of DNA that serve as protective ends to chromosomes, researchers have discovered. The findings show that this manipulation of telomeres may explain how viruses like herpes are able to successfully replicate while also revealing more about the protective role that telomeres play against other viruses.

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