Scientists make important step toward stopping plaque-like formations in Huntington's disease

Researchers describe a laboratory test that allows scientists to evaluate large numbers of fruit fly genes for a possible role in the formation of plaque-like protein aggregates within cells. Those genes often have counterparts in humans, which might then be manipulated to stop or slow the formation of plaque-like protein aggregates, the hallmark of Huntington's and other neurodegenerative diseases.

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